{
  "$type": "site.standard.document",
  "bskyPostRef": {
    "cid": "bafyreigmvzvmgkhl3wskfuru35heqxsqbz5cwnl2k6ebpi6fq373wm6bqy",
    "uri": "at://did:plc:ox2kiwcr2xmn32p25g472pp7/app.bsky.feed.post/3mk2bmerox5w2"
  },
  "coverImage": {
    "$type": "blob",
    "ref": {
      "$link": "bafkreifyai6h24mylqgvjhjc4mzi5wjcaibk3zdp35adg752ovawxdqwpu"
    },
    "mimeType": "image/webp",
    "size": 138250
  },
  "path": "/releases/2026/04/260420014744.htm",
  "publishedAt": "2026-04-20T07:14:27.000Z",
  "site": "https://www.sciencedaily.com",
  "textContent": "Cancer cells are known for their “glutamine addiction,” but many can escape this weakness by switching to alternative fuels. Researchers found that vitamin B7 acts like a metabolic “license,” enabling this escape route through a key enzyme. Without biotin, cancer cells lose that flexibility and stop growing. Mutations in a cancer-linked gene can make this vulnerability even stronger, offering a promising new target for therapy.",
  "title": "This missing vitamin could stop cancer cells in their tracks"
}